Oxalate-rich greens (spinach, beet greens, chard) cause kidney stones; each 100 mg of dietary oxalate raises urinary oxalate by about 2.7 mg.
Original statement
No Arabic original wording available.
Translation
Oxalate-rich greens (spinach, beet greens, chard) cause kidney stones; each 100 mg of dietary oxalate raises urinary oxalate by about 2.7 mg.
Origin
- Source status
- Archive editors
- Attribution
- Archive editors
- Origin
- https://eltayebat.com/rule/leafy-greens-cucurbits/
- Attribution uncertainty
- Archive editorial text and study selection written by the archive operators, not necessarily by Elawady.
- Claimed mechanism
Dietary oxalate increases urinary oxalate and calcium-oxalate crystallisation.
- Claimed health effect
Kidney stones.
- Population concerned
Everyone
- Geographic restriction by Elawady
none found
Scientific assessment
Mostly false – Essential parts of the statement are not supported by the evidence or are strongly exaggerated.
Certainty of evidence: moderate – Further research may still affect the assessment.
1. What was claimed?
Oxalate-rich greens cause kidney stones.
2. What is demonstrably correct?
Spinach is the main oxalate source; high spinach intake (≥8 servings/month) was modestly associated with stones in men (RR 1.30) and older women (RR 1.34). Dietary oxalate contributes to urinary oxalate (Crivelli 2020).
3. What is wrong or exaggerated?
The cohort authors concluded that the data 'do not implicate dietary oxalate as a major risk factor'. A vegetable- and legume-rich DASH diet was associated with markedly fewer stones (RR 0.55-0.60). The archive's cited J Urol study is an animal model with an oxalate precursor, not green vegetables; the '2.7 mg per 100 mg' figure could not be verified from the cited source.
4. What remains unclear?
The effect of very high spinach intake in specific populations (e.g. hot climates) is uncertain.
5. Which studies support it?
Khan 2007 (rat model); Taylor 2007 (modest association).
6. Which studies contradict it?
Taylor 2007 (conclusion); Taylor 2009 (DASH).
7. What applies to Germany and Europe?
Kidney-stone guidance in Europe advises adequate fluid and calcium, with oxalate moderation for recurrent stone formers.
8. What is known about Egypt?
Kidney stones are common in Egypt; hydration is the key preventive measure (not reviewed in detail).
9. What practical conclusion is appropriate?
For most people, eating greens does not meaningfully increase stone risk; stone formers should get individual advice.
Assessment details
- Rationale for certainty of evidence
Large cohorts (240,000 participants) found only a modest association between oxalate intake and stones in men and older women (RR ~1.2), none in younger women; the authors concluded that dietary oxalate is not a major risk factor. A DASH-style diet rich in vegetables and legumes was associated with 40-45% lower stone risk.
- Source quality
The archive's 2.7 mg figure does not appear in the abstract of the cited J Urol paper, which is a rat study using hydroxy-L-proline, not a human dietary-oxalate study.
- Regional transferability
US cohorts; transferable to Germany and Egypt with caution (Egypt has high stone prevalence linked to climate and fluid intake; not reviewed).
- Individual exceptions
Recurrent calcium-oxalate stone formers, people with enteric hyperoxaluria or CKD may need to limit high-oxalate foods individually and ensure adequate calcium.
- Regional scope of the claim
- general (biological/medical)
- Last reviewed
- 25 October 2026 provisional
In plain language
Spinach and some other greens contain oxalate, a substance found in most kidney stones. In large studies, people who ate the most oxalate had only a slightly higher risk of stones, and the authors concluded that dietary oxalate is not a major risk factor. People eating a diet rich in vegetables, fruit and legumes actually had far fewer kidney stones. The animal study cited by the archive does not test vegetables.
Studies on this claim
Studies supporting the claim (1)
-
Oxalate precursor and kidney crystals in rats
Khan SR et al.. J Urol 2007 · PMID 17870111 · DOI 10.1016/j.juro.2007.06.046Animal studycited by the Tayebat archiveabstract only checkedRats fed an oxalate precursor developed kidney crystals that partly cleared after stopping it.
Does the study support the specific claim? Unclear · Methodological quality: low · Certainty of evidence: see overall assessment of the claim
Original publication · Scientific details
Study explained simply
- What did the researchers want to find out?
Test oxalate reduction in rats
- How was the study done?
Animal experiment
- How many people, animals or samples were studied?
Rats
- What was actually found?
Crystals formed, partly reversible
- How large was the observed effect?
Not applicable
- What do the results mean in everyday life?
No direct meaning for vegetable eating.
- What are the study's limitations?
Animal; precursor
- What does it say about Elawady's specific claim?
Very indirect
- Do the results apply to Germany and Europe?
Low
- What can explicitly not be concluded from this study?
Cannot show greens cause stones in humans.
No study in this group is marked as key.
Studies contradicting or limiting the claim (2)
-
Dietary oxalate and kidney stones
Taylor EN, Curhan GC. J Am Soc Nephrol 2007 · PMID 17538185 · DOI 10.1681/ASN.2007020219Cohort studyindependent cross-checkabstract only checked★ key studyHighest oxalate intake raised stone risk only modestly (about 20%) in some groups; authors: not a major risk factor.
Does the study support the specific claim? No · Methodological quality: high · Certainty of evidence: see overall assessment of the claim
Original publication · Scientific details
Study explained simply
- What did the researchers want to find out?
Oxalate and stones
- How was the study done?
Cohorts
- How many people, animals or samples were studied?
240,681
- What was actually found?
Modest or no association
- How large was the observed effect?
RR ~1.2
- What do the results mean in everyday life?
Greens not a major stone risk.
- What are the study's limitations?
FFQ
- What does it say about Elawady's specific claim?
Contradicts general claim
- Do the results apply to Germany and Europe?
Moderate
- What can explicitly not be concluded from this study?
Cannot address stone formers specifically.
-
Vegetable-rich diet and kidney stones
Taylor EN et al.. J Am Soc Nephrol 2009 · PMID 19679672 · DOI 10.1681/ASN.2009030276Cohort studyindependent cross-checkabstract only checked★ key studyPeople with a diet rich in vegetables, fruit and legumes had about 40-45% fewer kidney stones.
Does the study support the specific claim? No · Methodological quality: high · Certainty of evidence: see overall assessment of the claim
Original publication · Scientific details
Study explained simply
- What did the researchers want to find out?
DASH and stones
- How was the study done?
Cohorts
- How many people, animals or samples were studied?
~240,000
- What was actually found?
Fewer stones
- How large was the observed effect?
RR 0.55-0.60
- What do the results mean in everyday life?
Vegetables protect rather than harm.
- What are the study's limitations?
Observational
- What does it say about Elawady's specific claim?
Contradicts claim
- Do the results apply to Germany and Europe?
Moderate
- What can explicitly not be concluded from this study?
Cannot prove causation.
No study in this group is marked as key.
Studies with mixed or unclear results (1)
-
Where urinary oxalate comes from
Crivelli JJ et al.. Nutrients 2020 · PMID 33379176 · DOI 10.3390/nu13010062Narrative reviewindependent cross-checkabstract only checkedDietary oxalate contributes to urinary oxalate, modified by gut bacteria and calcium.
Does the study support the specific claim? Partly · Methodological quality: moderate · Certainty of evidence: see overall assessment of the claim
Original publication · Scientific details
Study explained simply
- What did the researchers want to find out?
Review oxalate
- How was the study done?
Review
- How many people, animals or samples were studied?
Not applicable
- What was actually found?
Diet contributes; gut bacteria modify
- How large was the observed effect?
Not applicable
- What do the results mean in everyday life?
Calcium with meals and moderation help stone formers.
- What are the study's limitations?
Narrative
- What does it say about Elawady's specific claim?
Context
- Do the results apply to Germany and Europe?
High
- What can explicitly not be concluded from this study?
Cannot quantify stone risk.
No study in this group is marked as key.
Regional context
Germany / EU
Kidney-stone guidance in Europe advises adequate fluid and calcium, with oxalate moderation for recurrent stone formers.
Egypt
Kidney stones are common in Egypt; hydration is the key preventive measure (not reviewed in detail).
What applies in my country?
General scientific assessment
Mostly false Greens do not generally cause kidney stones; individual advice applies to stone formers.
The following areas of food safety are relevant to this statement:
- Pesticides and pesticide residues
- Contaminants (heavy metals, PFAS, PCBs, dioxins, nitrate)
- Pathogens (Salmonella, Campylobacter, etc.)
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References
- Dietary oxalate and calcium oxalate nephrolithiasis.. Khan SR et al.. J Urol 2007 · PMID 17870111 · DOI 10.1016/j.juro.2007.06.046
- Oxalate intake and the risk for nephrolithiasis.. Taylor EN, Curhan GC. J Am Soc Nephrol 2007 · PMID 17538185 · DOI 10.1681/ASN.2007020219
- DASH-style diet associates with reduced risk for kidney stones.. Taylor EN et al.. J Am Soc Nephrol 2009 · PMID 19679672 · DOI 10.1681/ASN.2009030276
- Contribution of Dietary Oxalate and Oxalate Precursors to Urinary Oxalate Excretion.. Crivelli JJ et al.. Nutrients 2020 · PMID 33379176 · DOI 10.3390/nu13010062